Aortic Pressures, Stiffness and Left Ventricular Function in Coronary Slow Flow Phenomenon

dc.authorscopusid55710786700
dc.authorscopusid6603279732
dc.authorscopusid41261948900
dc.authorscopusid23101274300
dc.authorscopusid35740159200
dc.authorscopusid6505680586
dc.authorwosidAlpsoy, Seref/Z-1808-2018
dc.contributor.authorTanrıverdi, Halil
dc.contributor.authorEvrengül, Harun
dc.contributor.authorKılıç, I. Doğu
dc.contributor.authorTaşköylü, Özgür
dc.contributor.authorDoğan, Güllü
dc.contributor.authorAlpsoy, Şeref
dc.date.accessioned2022-05-11T14:40:06Z
dc.date.available2022-05-11T14:40:06Z
dc.date.issued2010
dc.departmentFakülteler, Tıp Fakültesi, Dahili Tıp Bilimleri Bölümü, Kardiyoloji Ana Bilim Dalı
dc.description.abstractBackground: Coronary slow flow (CSF) phenomenon is characterized by delayed opacification of coronary vessels in a normal coronary angiogram. Although clinical and pathological features have been previously described, the underlying pathophysiology has not been fully elucidated. In this study, we aimed to evaluate central aortic pressure, aortic stiffness and left ventricular (LV) function in patients with coronary slow flow. Method and Results: The study population consisted of 154 patients who underwent coronary angiography because of typical and quasi-typical symptoms of angina. 81 patients with angiographically proven CSF and 73 cases with normal coronary flow pattern with similar risk profiles were enrolled in the study. Aortic pressures and indexes of elastic properties were evaluated using invasive methods. Aortic pulse pressure (mm Hg) and pulsality index of patients with CSF were found to be significantly higher than those of controls (60.8 +/- 13.8 vs. 53.7 +/- 14.5, p = 0.002; 0.84 +/- 0.22 vs. 0.67 +/- 0.18, p = 0.0001, respectively). LV Doppler parameters were deteriorated in patients with CSF. Aortic fractional pulse pressure and pulsality index were significantly correlated with the mean TIMI frame count and LV diastolic parameters in correlation analysis. Conclusion: The present findings allow us to conclude that impaired aortic elasticity in patients with CSF may also be responsible for the impaired LV diastolic parameters. Copyright (C) 2010 S. Karger AG, Basel
dc.identifier.doi10.1159/000316652
dc.identifier.endpage267
dc.identifier.issn0008-6312
dc.identifier.issn1421-9751
dc.identifier.issue4en_US
dc.identifier.pmid20798537
dc.identifier.scopus2-s2.0-77956072659
dc.identifier.scopusqualityQ3
dc.identifier.startpage261
dc.identifier.urihttps://doi.org/10.1159/000316652
dc.identifier.urihttps://hdl.handle.net/20.500.11776/8864
dc.identifier.volume116
dc.identifier.wosWOS:000283036900007
dc.identifier.wosqualityQ3
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.institutionauthorAlpsoy, Şeref
dc.language.isoen
dc.publisherKarger
dc.relation.ispartofCardiology
dc.relation.publicationcategoryDiğeren_US
dc.rightsinfo:eu-repo/semantics/closedAccess
dc.subjectAortic stiffness
dc.subjectCoronary slow flow
dc.subjectDiastolic dysfunction
dc.subjectPulse pressure
dc.subjectPulse-Wave Velocity
dc.subjectCardiovascular Mortality
dc.subjectIndependent Predictor
dc.subjectHypertensive Patients
dc.subjectEndothelial Function
dc.subjectDiastolic Function
dc.subjectArtery-Disease
dc.subjectAtherosclerosis
dc.subjectPerfusion
dc.subjectDoppler
dc.titleAortic Pressures, Stiffness and Left Ventricular Function in Coronary Slow Flow Phenomenon
dc.typeEditorial

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